http://www.eurekalert.org/pub_releases/2014-05/uoc-gdr050714.php
Gluten-free diet reduces risk of type 1 diabetes in mice
Gluten-free diet reduces risk of type 1 diabetes in mice
Verschrompelde nier en diabetes type 1
- Onderwerp starter Jur
- Startdatum
Blijkt dat diabetes type I kan samenhangen met geblokkeerde aderen waardoor de insuline niet in het bloed kan komen. Dit kan verklaren waarom men heeft uitgevonden dat diabetes type I vaak niet alleen verklaard kan worden vanuit de theorie dat de pancreas geen insuline meer aanmaakt. Serrapeptase kan helpen bij de aderen deblokkeren.
Quote:
Do a search for Dr Wong. He has 2 people who have got off insulin using Zymessence apparently. Don't know if this is true though.
I have also read that the tiny veins that are attached to the islets become blocked, and that the pancreas never stops producing insulin, but the veins become blocked and stop the insulin getting out. Serrapeptase is an enzyme that supposedly unblocks the bodies arteries and veins, and that this is why Zymessence can work in some individuals. I haven't tried it myself as yet, as i have been using a different kind of Serrapeptase bought from Good Health Naturally, here in the UK, but so far it hasn't worked. The product is called Serranol which HSI did an article about saying that it is the only version of serrapeptase that uses 100% serrapeptase. All others use a 14% blend.
I am going to try the Zymessence afterwards, it can't harm to try it. The 2 testimonials are on the Dr Wong website.
http://www.greenmedinfo.com/blog/6-bodily-tissues-can-be-regenerated-through-nutrition?page=2
Deze dame www.youtube.com/watch?v=DIYdYX0U3PM kwam aan nadat ze insuline ging spuiten na de diagnose diabetes type I net als de zus van Tralala (zie vorige pagina).
De lichamelijke conditie van zowel diabetes type I als II patienten verbetert na behandeling met ozontheraie https://www.fatsforum.nl/topic/ozontherapie
Quote:
In Russia, there was a treatment of ozone therapy in diabetes. In this treatment, 38 patients were asked to give their opinions. There were 20 people who were suffering from 1st type of diabetes and the rest from 2nd type with different degrees of severity. Most of these patients were at the decomposition state and 22 have serious complexities. The treatment included the use of intravenous drop injection of the physiological solution along with the rectum injection O3/O2 of the mixture. In this course, there were almost 7 to 10 treatments. The amazing thing was that the patients found considerable improvement in their health state. They stopped complaints about being thirsty, frequent urination, skin itching, dry in a mouth tingling sensation etc. During the first hours, the glucose level of the patients lowered to 50 percent of their original level. This was a huge success.In Russia, there was a treatment of ozone therapy in diabetes. In this treatment, 38 patients were asked to give their opinions. There were 20 people who were suffering from 1st type of diabetes and the rest from 2nd type with different degrees of severity. Most of these patients were at the decomposition state and 22 have serious complexities. The treatment included the use of intravenous drop injection of the physiological solution along with the rectum injection O3/O2 of the mixture. In this course, there were almost 7 to 10 treatments. The amazing thing was that the patients found considerable improvement in their health state. They stopped complaints about being thirsty, frequent urination, skin itching, dry in a mouth tingling sensation etc. During the first hours, the glucose level of the patients lowered to 50 percent of their original level. This was a huge success. And by the time the course ended, the glucose level lowered to almost 30 percent. The best news was that almost all the patients returned home with an improved health condition. And by the time the course ended, the glucose level lowered to almost 30 percent. The best news was that almost all the patients returned home with an improved health condition.
http://www.ozonehospital.com/ozone-therapy-for-diabetis.html
Quote:
Do a search for Dr Wong. He has 2 people who have got off insulin using Zymessence apparently. Don't know if this is true though.
I have also read that the tiny veins that are attached to the islets become blocked, and that the pancreas never stops producing insulin, but the veins become blocked and stop the insulin getting out. Serrapeptase is an enzyme that supposedly unblocks the bodies arteries and veins, and that this is why Zymessence can work in some individuals. I haven't tried it myself as yet, as i have been using a different kind of Serrapeptase bought from Good Health Naturally, here in the UK, but so far it hasn't worked. The product is called Serranol which HSI did an article about saying that it is the only version of serrapeptase that uses 100% serrapeptase. All others use a 14% blend.
I am going to try the Zymessence afterwards, it can't harm to try it. The 2 testimonials are on the Dr Wong website.
http://www.greenmedinfo.com/blog/6-bodily-tissues-can-be-regenerated-through-nutrition?page=2
Deze dame www.youtube.com/watch?v=DIYdYX0U3PM kwam aan nadat ze insuline ging spuiten na de diagnose diabetes type I net als de zus van Tralala (zie vorige pagina).
De lichamelijke conditie van zowel diabetes type I als II patienten verbetert na behandeling met ozontheraie https://www.fatsforum.nl/topic/ozontherapie
Quote:
In Russia, there was a treatment of ozone therapy in diabetes. In this treatment, 38 patients were asked to give their opinions. There were 20 people who were suffering from 1st type of diabetes and the rest from 2nd type with different degrees of severity. Most of these patients were at the decomposition state and 22 have serious complexities. The treatment included the use of intravenous drop injection of the physiological solution along with the rectum injection O3/O2 of the mixture. In this course, there were almost 7 to 10 treatments. The amazing thing was that the patients found considerable improvement in their health state. They stopped complaints about being thirsty, frequent urination, skin itching, dry in a mouth tingling sensation etc. During the first hours, the glucose level of the patients lowered to 50 percent of their original level. This was a huge success.In Russia, there was a treatment of ozone therapy in diabetes. In this treatment, 38 patients were asked to give their opinions. There were 20 people who were suffering from 1st type of diabetes and the rest from 2nd type with different degrees of severity. Most of these patients were at the decomposition state and 22 have serious complexities. The treatment included the use of intravenous drop injection of the physiological solution along with the rectum injection O3/O2 of the mixture. In this course, there were almost 7 to 10 treatments. The amazing thing was that the patients found considerable improvement in their health state. They stopped complaints about being thirsty, frequent urination, skin itching, dry in a mouth tingling sensation etc. During the first hours, the glucose level of the patients lowered to 50 percent of their original level. This was a huge success. And by the time the course ended, the glucose level lowered to almost 30 percent. The best news was that almost all the patients returned home with an improved health condition. And by the time the course ended, the glucose level lowered to almost 30 percent. The best news was that almost all the patients returned home with an improved health condition.
http://www.ozonehospital.com/ozone-therapy-for-diabetis.html
Quote:
Niacinamide has three major benefits for diabetics. First, it reduces nitric oxide synthase which helps retard beta cell death. Beta cells are the pancreatic cells that
produce insulin. Second, it enhances ox/redox function through restoration of NAD levels which help prevent cellular damage and improve regeneration. Third, it reduces glycosylated hemoglobin levels which reduces the peripheral organ and blood vessel oxidation load and damage from sugar metabolites. In type I diabetics, niacinamide supplementation may slow down pancreatic beta cell destruction (where insulin is formed) and enhance the cells’ regeneration. Niacinamide suppresses ADP-ribosylation reactions in beta cells as well as in immune cells and the endothelium. Cell death pathways and gene expression patterns are modified with high niacinamide doses leading to improved beta cell survival and an enhanced immunoregulatory balance (Kolb, Burkhart 1999). In another earlier study, nicotinamide supplementation was shown to result in lower glycosylated hemoglobin along with lower insulin doses. The authors concluded that niacinamide may be successful in improving metabolic control in recent onset type I diabetes, probably by increasing residual islet B-cell (beta cell) function (Pozzilli, et.al. 1989). Complete remissions of young adult type I diabetics lasting over two years are rare, yet a 1987 French study found two complete remissions out of seven patients given 3gm/day. They also found the mean dose of insulin was lower in the
niacinamide-treated group than the placebo group. (Vague, et.al 1987)
Niacinamide may also be of benefit in type II adult onset diabetes and chronic pancreatitis. Researchers observed improved glycemic parameters and other benefits by
the addition of niacinamide to nutritional therapies of type II diabetics. It is reasoned that reduction of glycosylated hemoglobin and enhancement of beta cell survival and function is beneficial in Type II diabetes as well. In a study of alcoholics with chronic pancreatitis, niacinamide significantly increased basal secretion of insulin, slightly improved glucose-stimulated secretion and promoted reduction of hypercoagulation and time to remission (Loginov et.al. 1999). Niacinamide may also retard nephropathy in diabetics and reduce liver toxicity in drug therapies (Kroger, et. al. 1999).
While high doses of B3 as nicotinic acid have been reported to worsen glycemic control, niacinamide’s positive effect on reducing glycosylated hemoglobin should be seriously considered. One to three grams of niacinamide per day is safely achievable. I have had only positive responses in my patients who use a multinutrient formula with large amounts of niacinamide.
In addition, niacinamide works synergistically with other supplements such as vitamin E, calcium-AEP, chromium picolinate, vanadyl sulfate, lipoic acid and the herb Goat’s
Rue (Galega officinalis) to reduce blood sugar and glycosylated hemoglobin, and regulate blood insulin. Studies show that control of glycosylated hemoglobin and insulin are key factors in management of Type I and II diabetes because both contribute heavily to the deleterious effects of the disease process.
Dietary management is extremely important in the maintenance of glycemic control. Many Type II adult onset diabetics do not make adequate diet and lifestyle modifications and continue with dietary habits that contributed to their condition in the first place. When supplementing over 500mg/day of niacinamide, it is recommended that diabetics (both Type I and II) monitor not only blood sugar, but also glycosylated hemoglobin and insulin levels. They can then adjust their individual niacinamide dosing and diet accordingly to achieve an optimum reduction in glycosylated hemoglobin.
http://www.vrp.com/single-vitamins/...another-look-at-clinical-uses-for-niacinamide zie ook: https://www.fatsforum.nl/topic/niacin-flush-en-insulineresistentie
Niacinamide has three major benefits for diabetics. First, it reduces nitric oxide synthase which helps retard beta cell death. Beta cells are the pancreatic cells that
produce insulin. Second, it enhances ox/redox function through restoration of NAD levels which help prevent cellular damage and improve regeneration. Third, it reduces glycosylated hemoglobin levels which reduces the peripheral organ and blood vessel oxidation load and damage from sugar metabolites. In type I diabetics, niacinamide supplementation may slow down pancreatic beta cell destruction (where insulin is formed) and enhance the cells’ regeneration. Niacinamide suppresses ADP-ribosylation reactions in beta cells as well as in immune cells and the endothelium. Cell death pathways and gene expression patterns are modified with high niacinamide doses leading to improved beta cell survival and an enhanced immunoregulatory balance (Kolb, Burkhart 1999). In another earlier study, nicotinamide supplementation was shown to result in lower glycosylated hemoglobin along with lower insulin doses. The authors concluded that niacinamide may be successful in improving metabolic control in recent onset type I diabetes, probably by increasing residual islet B-cell (beta cell) function (Pozzilli, et.al. 1989). Complete remissions of young adult type I diabetics lasting over two years are rare, yet a 1987 French study found two complete remissions out of seven patients given 3gm/day. They also found the mean dose of insulin was lower in the
niacinamide-treated group than the placebo group. (Vague, et.al 1987)
Niacinamide may also be of benefit in type II adult onset diabetes and chronic pancreatitis. Researchers observed improved glycemic parameters and other benefits by
the addition of niacinamide to nutritional therapies of type II diabetics. It is reasoned that reduction of glycosylated hemoglobin and enhancement of beta cell survival and function is beneficial in Type II diabetes as well. In a study of alcoholics with chronic pancreatitis, niacinamide significantly increased basal secretion of insulin, slightly improved glucose-stimulated secretion and promoted reduction of hypercoagulation and time to remission (Loginov et.al. 1999). Niacinamide may also retard nephropathy in diabetics and reduce liver toxicity in drug therapies (Kroger, et. al. 1999).
While high doses of B3 as nicotinic acid have been reported to worsen glycemic control, niacinamide’s positive effect on reducing glycosylated hemoglobin should be seriously considered. One to three grams of niacinamide per day is safely achievable. I have had only positive responses in my patients who use a multinutrient formula with large amounts of niacinamide.
In addition, niacinamide works synergistically with other supplements such as vitamin E, calcium-AEP, chromium picolinate, vanadyl sulfate, lipoic acid and the herb Goat’s
Rue (Galega officinalis) to reduce blood sugar and glycosylated hemoglobin, and regulate blood insulin. Studies show that control of glycosylated hemoglobin and insulin are key factors in management of Type I and II diabetes because both contribute heavily to the deleterious effects of the disease process.
Dietary management is extremely important in the maintenance of glycemic control. Many Type II adult onset diabetics do not make adequate diet and lifestyle modifications and continue with dietary habits that contributed to their condition in the first place. When supplementing over 500mg/day of niacinamide, it is recommended that diabetics (both Type I and II) monitor not only blood sugar, but also glycosylated hemoglobin and insulin levels. They can then adjust their individual niacinamide dosing and diet accordingly to achieve an optimum reduction in glycosylated hemoglobin.
http://www.vrp.com/single-vitamins/...another-look-at-clinical-uses-for-niacinamide zie ook: https://www.fatsforum.nl/topic/niacin-flush-en-insulineresistentie
Ik sprak zojuist een vriendin (20 jaar) met diabetes type 1. Ze weet sinds 2,5 jaar dat ze de ziekte onder de leden heeft, maar heeft niet geïnvesteerd in een gezond(er) voedingspatroon. Eigenlijk wil ze alles blijven eten wat ze al deed (boterhammen als ontbijt, af en toe taart of friet), het enige verschil is dat ze vaak suikerwaarde moet meten en insuline moet spuiten. Dat geeft al zoveel gedoe dat ze niet 'ook nog eens rekening wil houden met bijvoorbeeld welke vetten (on)gezond zijn'. Dan moet je namelijk nieuwe berekeningen in je hoofd maken, aangezien de combi vetten+koolhydraten anders werken dan alleen koolhydraten (suikerpiek én langzame energie van vet, klopt dat?).
Ze wil denk ik niet snel veranderen hierin: ze kan lekker alles blijven eten, zolang ze maar insuline spuit.
Mijn vraag: welk belang hebben diabetici type 1 bij een constante bloedsuikerspiegel? Ze zijn 'toch al ziek' en insuline is er genoeg... Waarom lopen ze dan meer risico op hart- en vaatziekten (als dit zo is)?
Ze wil denk ik niet snel veranderen hierin: ze kan lekker alles blijven eten, zolang ze maar insuline spuit.
Mijn vraag: welk belang hebben diabetici type 1 bij een constante bloedsuikerspiegel? Ze zijn 'toch al ziek' en insuline is er genoeg... Waarom lopen ze dan meer risico op hart- en vaatziekten (als dit zo is)?
ad ligtvoet
New member
Een belangrijke vraag is of de hoeveelheid insuline die gespoten wordt ook voldoende is om de alfa cellen te stoppen met het produceren van glucagon. Zo niet, dan blijft het lichaam glucose produceren/vrijgeven. Dit is een rede waarom diabetici steeds zieker worden en het glucose niveau moeilijk constant te houden is.
De bloedsuikerspiegel moet niet perse constant zijn maar wel laag genoeg anders krijg je dus deze problemen als har-t en vaat ziekten. Het constant(op een bepaald 'gezond 'niveau) houden is praktischer voor een diabeet lijkt mij.
Een hoop gedoe, zoals gezegd werd, weegt niet op tegen......(flat line).
ad
De bloedsuikerspiegel moet niet perse constant zijn maar wel laag genoeg anders krijg je dus deze problemen als har-t en vaat ziekten. Het constant(op een bepaald 'gezond 'niveau) houden is praktischer voor een diabeet lijkt mij.
Een hoop gedoe, zoals gezegd werd, weegt niet op tegen......(flat line).
ad
Ik kan je niet helemaal volgen. Glucagon voorkomt een te lage bloedsuikerspiegel, dat is toch niet schadelijk? En door insuline te spuiten verminder je toch niet glucagon?
Een meer constante bloedsuikerspiegel zonder te hoge waarden is belangrijk om diabetes te voorkomen. Maar wat is er mis met een hoge koolhydraatconsumptie als je, als diabeet zijnde, altijd gewoon insuline kunt spuiten?
Een meer constante bloedsuikerspiegel zonder te hoge waarden is belangrijk om diabetes te voorkomen. Maar wat is er mis met een hoge koolhydraatconsumptie als je, als diabeet zijnde, altijd gewoon insuline kunt spuiten?
ad ligtvoet
New member
Eigen productie van insuline in een fysiologische gezonde hoeveelheid zal glucagon stoppen. Inderdaad glucagon kan een te lage spiegel voorkomen en ja, door insuline te spuiten verminder je dus de glucagon productie niet. Dus wanneer deze vrij blijft komen zal er ook constant glucose vrijkomen, hierdoor blijft de glucosespiegel te hoog. Meer insuline spuiten is dan de enige optie, maar dit is niet gezond.
Je kunt deze reeds bestaande diabetes niet meer voorkomen door een constante spiegel van glucose. We hebben het hier over diabetici 1.
Als ik het goed lees zie jij ook geen nadeel in hoge consumptie van KH, ongeacht eigen insuline productie of injecteren ervan.
AD
Je kunt deze reeds bestaande diabetes niet meer voorkomen door een constante spiegel van glucose. We hebben het hier over diabetici 1.
Als ik het goed lees zie jij ook geen nadeel in hoge consumptie van KH, ongeacht eigen insuline productie of injecteren ervan.
AD
Dank. Ik vond ook deze site erg verhelderend.
Ik zie veel nadelen in hoge KH-consumptie als je geen diabetes (1 of 2) hebt. Dat zijn we eens. Ik snapte alleen niet wat er mis mee is als je al diabetes type 1 hebt. Maar na dit citaat van bovenstaande site wel:
De toegediende injectie zorgt voor een fysiologisch idioot hoge insulineconcentratie in de periferie, terwijl het signaal in de eilandjes nog altijd veel te laag is om de glucagonproductie op normale wijze af te remmen, zoals bij gezonde personen gebeurt. De lever van een type 1 diabeet produceert dus bijna altijd te veel suiker en de arme patiënt kan die kraan niet dicht zetten. Het enige wat hij kan doen is zijn cellen met behulp van fysiologisch enorme hoeveelheden insuline overdreven veel glucose te laten opnemen. Na verloop van tijd zal al die insuline ook de alfacellen bereiken, waarna de glucagonproductie wel daalt, met potentieel fatale en in elk geval erg vervelende hypoglycaemie tot gevolg. Diabeten type 1 zijn gedwongen een spel te spelen dat ze niet kunnen winnen. De beste manier om de achtbaan enigszins onder controle te krijgen is een sterk koolhydraatbeperkt dieet.
Ik zie veel nadelen in hoge KH-consumptie als je geen diabetes (1 of 2) hebt. Dat zijn we eens. Ik snapte alleen niet wat er mis mee is als je al diabetes type 1 hebt. Maar na dit citaat van bovenstaande site wel:
De toegediende injectie zorgt voor een fysiologisch idioot hoge insulineconcentratie in de periferie, terwijl het signaal in de eilandjes nog altijd veel te laag is om de glucagonproductie op normale wijze af te remmen, zoals bij gezonde personen gebeurt. De lever van een type 1 diabeet produceert dus bijna altijd te veel suiker en de arme patiënt kan die kraan niet dicht zetten. Het enige wat hij kan doen is zijn cellen met behulp van fysiologisch enorme hoeveelheden insuline overdreven veel glucose te laten opnemen. Na verloop van tijd zal al die insuline ook de alfacellen bereiken, waarna de glucagonproductie wel daalt, met potentieel fatale en in elk geval erg vervelende hypoglycaemie tot gevolg. Diabeten type 1 zijn gedwongen een spel te spelen dat ze niet kunnen winnen. De beste manier om de achtbaan enigszins onder controle te krijgen is een sterk koolhydraatbeperkt dieet.
ad ligtvoet
New member
Ok, dat is je nu duidelijk.
wanneer je naar de gezondheid situatie van een diabeet kijkt en/of het verloop ervan bij diabeten over een langere periode dan zie je toch al genoeg. Dus dan zou er een belletje moeten gaan rinkelen.
Nu je vriendin nog overtuigen dat blind worden en amputaties niet zo fijn zijn. Sugarcoating the truth will not help.
ad
wanneer je naar de gezondheid situatie van een diabeet kijkt en/of het verloop ervan bij diabeten over een langere periode dan zie je toch al genoeg. Dus dan zou er een belletje moeten gaan rinkelen.
Nu je vriendin nog overtuigen dat blind worden en amputaties niet zo fijn zijn. Sugarcoating the truth will not help.
ad
Lievergezond
Well-known member
Destroyed Beta Cells
The main factor in type 1 diabetes is clearly the destruction of the insulin-producing beta cells in the pancreas, which is due to an inflammatory autoimmune reaction. Diabetes may manifest only after 90 per cent of the beta cells have been destroyed. The reason for this abnormal immune response is not well understood in medical circles.
Nevertheless, there are several important clues. Food allergy is one of them, dysbiosis or overgrowth of the intestines with pathogenic microbes another, while a third clue is vitamin B6 deficiency.
The diabetic pancreas shows a greatly increased number of white blood cells of a type associated with allergies. When type I diabetics are fasted in an appropriate way, their blood sugar levels often return to normal and may remain normal as long as only selected non-allergenic foods are used. With other foods, however, blood sugar levels may immediately go very high.
However, the question remains why an allergic reaction targets specifically the pancreas when usually other types of allergic reactions occur. It is here that the other two clues may provide the answer. When the normal protective gut bacteria are under stress, pathogenic microbes will take over. This is now very common but usually they remain further down towards the large intestine. Should they, however, invade the duodenum, the upper part of the small intestine, then pancreatitis or inflammation of the pancreas may result. This has been demonstrated experimentally. It is not even necessary for microbes to invade the pancreas itself as their breakdown products or endotoxins do most of the damage.
Clearly, a low-grade chronic inflammation of the pancreas makes it a primary target organ for any allergic reaction. Alternatively or in addition, vitamin B6 deficiency has been shown to damage the insulin-producing beta cells.
Even with a mild vitamin B6 deficiency, the amino acid tryptophan cannot be properly metabolised, part of which is normally converted to niacin or nicotinamide. Instead, an abnormal metabolite, xanthurenic acid, accumulates. High levels of this have been shown in animal experiments to damage the beta cells and within days such animals developed diabetes. The sooner the missing vitamin was supplied in high doses, the easier the blood sugar regulation could be normalised again.
Individuals who are even mildly deficient in vitamin B6 excrete xanthurenic acid in the urine. This is used as a laboratory test for vitamin B6 deficiency. Insulin-dependent diabetics generally excrete large amounts of xanthurenic acid, especially those with retinopathies (damaged retina). Magnesium and zinc supplements reduce the formation of xanthurenic acid. Both minerals are deficient in diabetics. A study found the diabetes death rate four times higher in areas with low water magnesium levels than in high magnesium areas.
Another interesting facet is that high doses of nicotinamide can postpone the need for insulin injections in newly diagnosed type I diabetics for months and even years. The explanation: a high level of this B vitamin in the blood inhibits the formation of xanthurenic acid from tryptophan in addition to protecting beta cells from autoimmune attack.
THE MILK AND GLUTEN CONNECTIONS
However, now comes the really important bit of information that ties together all of the foregoing parts. A study of several hundred newly diagnosed diabetic children revealed an immune response to a fragment of cows' milk protein in all of them. What is more, this protein fragment has the same composition as one called P69 on the beta cells.
Juvenile diabetes is much higher in those who have been bottle-fed rather than breast-fed, and it is lower in communities that consume fewer cows' milk products. However, it appears that this protein fragment is only a problem with milk from Friesian cows (called A1 milk) but not with milk from other, lower-yielding, breeds that produce A2 milk. Most of presently consumed milk is A1 milk.
P69 is usually protected inside the beta cells and comes only to the surface during microbial and especially viral infections. At that time the immune system can mistake it for cows' milk protein, attack it, and destroy the beta cell in the process. The problem is that bottle-fed infants are very susceptible to colds, respiratory and gastrointestinal infections. It is regarded as normal for them to have six and more infections a year, while these are rare with breast-fed infants.
But it does not end there. Bottle-fed infants also frequently receive antibiotics that then encourage overgrowth of the intestines with undesirable microbes, and a tendency to chronic pancreatitis. One type of E. coli bacteria is harmless in the large intestines but it has the potential for causing great damage in the small intestine. That is because it produces a molecule that is very similar to insulin. When the immune system becomes activated against this molecule, it may then also direct its attack against related features at the beta cells.
This shows that a combination of two factors is required to trigger an attack on the insulin-producing beta cells: one factor that brings P69 to the surface of the beta cells, and another factor that activates the immune system to attack them. As the first factor we may have high concentrations of xanthurenic acid or frequent colds of bottle-fed babies, and if antibiotics are used, then this also promotes overgrowth of the small intestines with pathogenic microbes, including E. coli as the second factor. This is reinforced when ingesting cow’s milk, which intensifies the attack on the beta cells.
More recently it has also been shown that an autoimmune attack on the pancreas can be triggered by a “leaky gut” or increased intestinal permeability. This has been shown to be triggered by gluten ingestion, especially from wheat.
Nitrosamine is a chemical especially high in preserved small goods, but it may also be formed from the high nitrate content of chemically fertilised produce. A significant association between nitrosamine and type I diabetes has been found, although it may only act indirectly by causing dysbiosis like so many other chemicals.
A Case History
My very first patient was a type 1 diabetic. Earlier I had worked mainly in medical university departments but at that time I was just writing with no intention of becoming a natural therapist. Unexpectedly a young man who was my friend was found to be diabetic when he was admitted to a hospital with symptoms close to diabetic coma. After being released with two daily insulin injections he asked me for help.
Immediately he stopped using insulin and went on an apple diet instead, followed by an organic raw-food diet including fermented goats' milk, but no vitamin or mineral supplements, herbs or other remedies. On the first day of this regimen his blood sugar level was 210 mg% or 11.8 mmol/l. Ten days later it was normal and remained normal even after the introduction of a normal mixed and cooked diet, and also the glucose tolerance test performed on the 24th day at the hospital was normal.
This diet was successful because it addressed the two most important factors in his condition: it eliminated foods and chemicals that have been allergenic or incompatible and it also sanitised the intestine by establishing a healthy micro-flora. However, the longer the condition is just 'managed' with insulin, the more of the beta cells become destroyed and the longer may be the road to recovery.
Nevertheless, even in advanced conditions it should frequently be possible to phase insulin out as a diet can be devised to regulate the blood sugar level. At the same time, with the elimination of the conditions that lead to the destruction of the beta cells, these have a chance to regenerate, with the internal blood sugar regulation becoming more effective in time so that the initially very strict diet can gradually been relaxed. http://www.health-science-spirit.com/diabetestruth.html
The Basic Diabetes 1 Diet
Children, depending on age, may remain for one to several days on this changeover cleansing routine, while adults may continue for one to two or three weeks, or until the blood sugar stabilises at an improved level. Start with one meal a day of the foods allowed in the basic diet. A day or two later increase this to two meals. If your blood sugar level is now reasonably low and stable without insulin, then start with allergy-testing the individual foods of the maintenance diet. If you still require insulin, remain for several weeks on the basic diet before you start allergy testing new foods.
The basic diet is a ‘safe’ diet with a minimum of glucose or foods such as starch that yield glucose during digestion. Suitable foods are meat, fish and other seafood, eggs, goats’ milk products (preferably fermented), Brazil nuts, ground linseed, avocados, lettuce and other leaf vegetables, celery, cucumber, some fresh tomato and raw onion for flavouring a salad, tart apples, lemon and lime juice. Green beans are especially good.
For normalising or stabilising the blood sugar level meat and fish may be conventionally cooked. However, the higher the cooking temperature, the greater will be the long-term health damage. Therefore, boil or steam but do not heat the food to above the boiling point. Nevertheless, much greater benefit in overall health will result from eating these foods predominantly raw. Use additional digestive enzymes with all cooked foods, but especially with cooked flesh foods.
Raw food and digestive enzymes are especially important for those who like to re-generate their insulin production, or for anyone with damage to their eyes, kidneys or blood vessels. Raw meat or fish are quite safe if you take suitable precautions. Try to find a source of organic meat and fowl, or otherwise grass-fed beef and free-range chicken. According to the Australian Department of Primary Industries freezing meat for about 10 days at minus 8°C or colder will kill any tapeworm cysts. Avoid any meat from feedlots, or fish from fish farms. Minimise eating large predatory fish, such as shark or tuna, because of their high mercury content. In addition use parasite remedies as outlined below.
Besides using raw minced meat, you may marinate diced meat or fish, or blend fish or chicken with water. Fresh fish often is sufficiently tender and tasty as it is. To marinate you immerse diced meat or fish in lemon juice or diluted cider vinegar and keep it refrigerated overnight.
Use raw onion, lime or lemon juice, ginger or chilli or any other herbs or spices for flavouring meals. If trying out raw flesh foods start using small amounts and increase gradually. Minimise the use of salt, and avoid it if you have a kidney problem, except if your blood pressure is rather low. Small amounts of magnesium chloride or potassium chloride are beneficial instead of salt. Egg yolk is best eaten raw and mixed with other food, while the white may be cooked; as a compromise you may soft-boil or lightly poach the egg, in any case the yolk should remain soft.
In addition to meat, fish or other seafood, egg or non-pasteurised goats’ cheese, eat a salad with each meal using the mentioned low-carbohydrate vegetables and possibly yoghurt. You may use virgin coconut oil and extra virgin olive oil. Avoid tinned fish with any kind of sauce, sausages or other manufactured meat.
Besides one or two meals, have apples or possibly pears and frequent drinks with diluted lime or lemon juice. Whenever you feel hungry, mix a teaspoon of spirulina (or chlorella) powder, a tablespoon of extra virgin olive oil, and a pinch of lecithin with water (warm or cold) or with diluted lime or lemon juice as an appetite suppressant and energiser.
Finally, several times a day before or between meals mix a teaspoon of psyllium hulls in a large glass of water and drink immediately. This is to help clean out the intestinal tract and ensure two to three easy bowel movements each day.
If your blood sugar level remains more or less normal, or after several weeks on this basic diet you may use allergy testing as described below to see how you react to additional foods as suggested for type 2 diabetics. Continue to use only non-reacting or safe foods. http://www.health-science-spirit.com/diabetestruth.html
The main factor in type 1 diabetes is clearly the destruction of the insulin-producing beta cells in the pancreas, which is due to an inflammatory autoimmune reaction. Diabetes may manifest only after 90 per cent of the beta cells have been destroyed. The reason for this abnormal immune response is not well understood in medical circles.
Nevertheless, there are several important clues. Food allergy is one of them, dysbiosis or overgrowth of the intestines with pathogenic microbes another, while a third clue is vitamin B6 deficiency.
The diabetic pancreas shows a greatly increased number of white blood cells of a type associated with allergies. When type I diabetics are fasted in an appropriate way, their blood sugar levels often return to normal and may remain normal as long as only selected non-allergenic foods are used. With other foods, however, blood sugar levels may immediately go very high.
However, the question remains why an allergic reaction targets specifically the pancreas when usually other types of allergic reactions occur. It is here that the other two clues may provide the answer. When the normal protective gut bacteria are under stress, pathogenic microbes will take over. This is now very common but usually they remain further down towards the large intestine. Should they, however, invade the duodenum, the upper part of the small intestine, then pancreatitis or inflammation of the pancreas may result. This has been demonstrated experimentally. It is not even necessary for microbes to invade the pancreas itself as their breakdown products or endotoxins do most of the damage.
Clearly, a low-grade chronic inflammation of the pancreas makes it a primary target organ for any allergic reaction. Alternatively or in addition, vitamin B6 deficiency has been shown to damage the insulin-producing beta cells.
Even with a mild vitamin B6 deficiency, the amino acid tryptophan cannot be properly metabolised, part of which is normally converted to niacin or nicotinamide. Instead, an abnormal metabolite, xanthurenic acid, accumulates. High levels of this have been shown in animal experiments to damage the beta cells and within days such animals developed diabetes. The sooner the missing vitamin was supplied in high doses, the easier the blood sugar regulation could be normalised again.
Individuals who are even mildly deficient in vitamin B6 excrete xanthurenic acid in the urine. This is used as a laboratory test for vitamin B6 deficiency. Insulin-dependent diabetics generally excrete large amounts of xanthurenic acid, especially those with retinopathies (damaged retina). Magnesium and zinc supplements reduce the formation of xanthurenic acid. Both minerals are deficient in diabetics. A study found the diabetes death rate four times higher in areas with low water magnesium levels than in high magnesium areas.
Another interesting facet is that high doses of nicotinamide can postpone the need for insulin injections in newly diagnosed type I diabetics for months and even years. The explanation: a high level of this B vitamin in the blood inhibits the formation of xanthurenic acid from tryptophan in addition to protecting beta cells from autoimmune attack.
THE MILK AND GLUTEN CONNECTIONS
However, now comes the really important bit of information that ties together all of the foregoing parts. A study of several hundred newly diagnosed diabetic children revealed an immune response to a fragment of cows' milk protein in all of them. What is more, this protein fragment has the same composition as one called P69 on the beta cells.
Juvenile diabetes is much higher in those who have been bottle-fed rather than breast-fed, and it is lower in communities that consume fewer cows' milk products. However, it appears that this protein fragment is only a problem with milk from Friesian cows (called A1 milk) but not with milk from other, lower-yielding, breeds that produce A2 milk. Most of presently consumed milk is A1 milk.
P69 is usually protected inside the beta cells and comes only to the surface during microbial and especially viral infections. At that time the immune system can mistake it for cows' milk protein, attack it, and destroy the beta cell in the process. The problem is that bottle-fed infants are very susceptible to colds, respiratory and gastrointestinal infections. It is regarded as normal for them to have six and more infections a year, while these are rare with breast-fed infants.
But it does not end there. Bottle-fed infants also frequently receive antibiotics that then encourage overgrowth of the intestines with undesirable microbes, and a tendency to chronic pancreatitis. One type of E. coli bacteria is harmless in the large intestines but it has the potential for causing great damage in the small intestine. That is because it produces a molecule that is very similar to insulin. When the immune system becomes activated against this molecule, it may then also direct its attack against related features at the beta cells.
This shows that a combination of two factors is required to trigger an attack on the insulin-producing beta cells: one factor that brings P69 to the surface of the beta cells, and another factor that activates the immune system to attack them. As the first factor we may have high concentrations of xanthurenic acid or frequent colds of bottle-fed babies, and if antibiotics are used, then this also promotes overgrowth of the small intestines with pathogenic microbes, including E. coli as the second factor. This is reinforced when ingesting cow’s milk, which intensifies the attack on the beta cells.
More recently it has also been shown that an autoimmune attack on the pancreas can be triggered by a “leaky gut” or increased intestinal permeability. This has been shown to be triggered by gluten ingestion, especially from wheat.
Nitrosamine is a chemical especially high in preserved small goods, but it may also be formed from the high nitrate content of chemically fertilised produce. A significant association between nitrosamine and type I diabetes has been found, although it may only act indirectly by causing dysbiosis like so many other chemicals.
A Case History
My very first patient was a type 1 diabetic. Earlier I had worked mainly in medical university departments but at that time I was just writing with no intention of becoming a natural therapist. Unexpectedly a young man who was my friend was found to be diabetic when he was admitted to a hospital with symptoms close to diabetic coma. After being released with two daily insulin injections he asked me for help.
Immediately he stopped using insulin and went on an apple diet instead, followed by an organic raw-food diet including fermented goats' milk, but no vitamin or mineral supplements, herbs or other remedies. On the first day of this regimen his blood sugar level was 210 mg% or 11.8 mmol/l. Ten days later it was normal and remained normal even after the introduction of a normal mixed and cooked diet, and also the glucose tolerance test performed on the 24th day at the hospital was normal.
This diet was successful because it addressed the two most important factors in his condition: it eliminated foods and chemicals that have been allergenic or incompatible and it also sanitised the intestine by establishing a healthy micro-flora. However, the longer the condition is just 'managed' with insulin, the more of the beta cells become destroyed and the longer may be the road to recovery.
Nevertheless, even in advanced conditions it should frequently be possible to phase insulin out as a diet can be devised to regulate the blood sugar level. At the same time, with the elimination of the conditions that lead to the destruction of the beta cells, these have a chance to regenerate, with the internal blood sugar regulation becoming more effective in time so that the initially very strict diet can gradually been relaxed. http://www.health-science-spirit.com/diabetestruth.html
The Basic Diabetes 1 Diet
Children, depending on age, may remain for one to several days on this changeover cleansing routine, while adults may continue for one to two or three weeks, or until the blood sugar stabilises at an improved level. Start with one meal a day of the foods allowed in the basic diet. A day or two later increase this to two meals. If your blood sugar level is now reasonably low and stable without insulin, then start with allergy-testing the individual foods of the maintenance diet. If you still require insulin, remain for several weeks on the basic diet before you start allergy testing new foods.
The basic diet is a ‘safe’ diet with a minimum of glucose or foods such as starch that yield glucose during digestion. Suitable foods are meat, fish and other seafood, eggs, goats’ milk products (preferably fermented), Brazil nuts, ground linseed, avocados, lettuce and other leaf vegetables, celery, cucumber, some fresh tomato and raw onion for flavouring a salad, tart apples, lemon and lime juice. Green beans are especially good.
For normalising or stabilising the blood sugar level meat and fish may be conventionally cooked. However, the higher the cooking temperature, the greater will be the long-term health damage. Therefore, boil or steam but do not heat the food to above the boiling point. Nevertheless, much greater benefit in overall health will result from eating these foods predominantly raw. Use additional digestive enzymes with all cooked foods, but especially with cooked flesh foods.
Raw food and digestive enzymes are especially important for those who like to re-generate their insulin production, or for anyone with damage to their eyes, kidneys or blood vessels. Raw meat or fish are quite safe if you take suitable precautions. Try to find a source of organic meat and fowl, or otherwise grass-fed beef and free-range chicken. According to the Australian Department of Primary Industries freezing meat for about 10 days at minus 8°C or colder will kill any tapeworm cysts. Avoid any meat from feedlots, or fish from fish farms. Minimise eating large predatory fish, such as shark or tuna, because of their high mercury content. In addition use parasite remedies as outlined below.
Besides using raw minced meat, you may marinate diced meat or fish, or blend fish or chicken with water. Fresh fish often is sufficiently tender and tasty as it is. To marinate you immerse diced meat or fish in lemon juice or diluted cider vinegar and keep it refrigerated overnight.
Use raw onion, lime or lemon juice, ginger or chilli or any other herbs or spices for flavouring meals. If trying out raw flesh foods start using small amounts and increase gradually. Minimise the use of salt, and avoid it if you have a kidney problem, except if your blood pressure is rather low. Small amounts of magnesium chloride or potassium chloride are beneficial instead of salt. Egg yolk is best eaten raw and mixed with other food, while the white may be cooked; as a compromise you may soft-boil or lightly poach the egg, in any case the yolk should remain soft.
In addition to meat, fish or other seafood, egg or non-pasteurised goats’ cheese, eat a salad with each meal using the mentioned low-carbohydrate vegetables and possibly yoghurt. You may use virgin coconut oil and extra virgin olive oil. Avoid tinned fish with any kind of sauce, sausages or other manufactured meat.
Besides one or two meals, have apples or possibly pears and frequent drinks with diluted lime or lemon juice. Whenever you feel hungry, mix a teaspoon of spirulina (or chlorella) powder, a tablespoon of extra virgin olive oil, and a pinch of lecithin with water (warm or cold) or with diluted lime or lemon juice as an appetite suppressant and energiser.
Finally, several times a day before or between meals mix a teaspoon of psyllium hulls in a large glass of water and drink immediately. This is to help clean out the intestinal tract and ensure two to three easy bowel movements each day.
If your blood sugar level remains more or less normal, or after several weeks on this basic diet you may use allergy testing as described below to see how you react to additional foods as suggested for type 2 diabetics. Continue to use only non-reacting or safe foods. http://www.health-science-spirit.com/diabetestruth.html
Lievergezond
Well-known member
Beta-Cell Regeneration – Unfortunately, the medical community has yet to harness the diabetes-reversing potential of natural compounds. Whereas expensive stem cell therapies, islet cell transplants, and an array of synthetic drugs in the developmental pipeline are the focus of billions of dollars of research, annually, our kitchen cupboards and backyards may already contain the long sought-after cure for type 1 diabetes. The following compounds have been demonstrated experimentally to regenerate the insulin-producing beta cells, which are destroyed in insulin dependent diabetes, and which once restored, may (at least in theory) restore the health of the patient to the point where they no longer require insulin replacement.
Gymenna Sylvestre (“the sugar destroyer”)
Nigella Sativa (“black cumin”)
Vitamin D
Curcumin (from the spice Turmeric)
Arginine
Avocado
Berberine (found in bitter herbs such as Goldenseal and Barberry)
Bitter Melon
Chard (yes, the green leafy vegetables)
Corn Silk
Stevia
Sulforaphane (especially concentrated in broccoli sprouts)
[view the first-hand biomedical citations on the Beta Cell Regeneration research page] http://www.greenmedinfo.com/keyword/beta-cell-regeneration http://www.greenmedinfo.com/blog/6-bodily-tissues-can-be-regenerated-through-nutrition
Gymenna Sylvestre (“the sugar destroyer”)
Nigella Sativa (“black cumin”)
Vitamin D
Curcumin (from the spice Turmeric)
Arginine
Avocado
Berberine (found in bitter herbs such as Goldenseal and Barberry)
Bitter Melon
Chard (yes, the green leafy vegetables)
Corn Silk
Stevia
Sulforaphane (especially concentrated in broccoli sprouts)
[view the first-hand biomedical citations on the Beta Cell Regeneration research page] http://www.greenmedinfo.com/keyword/beta-cell-regeneration http://www.greenmedinfo.com/blog/6-bodily-tissues-can-be-regenerated-through-nutrition
Forum statistieken
