Paradogma lees dit maar is door:
https://www.fatsforum.nl/topic/a1-en-a2-melk-en-caseine
https://www.fatsforum.nl/topic/rich...imal-heeft-nu-ook-rauwe-melk-en-kefir-ontdekt
https://www.fatsforum.nl/topic/oppassen-met-koemelk-ook-rauwe
Lees de hier volgende maar is goed na:
Update: I neglected to address the A1 vs. A2 issue with milk. Here's a study.
This review outlines a hypothesis that A1 one of the common variants of beta-casein, a major protein in cows milk could facilitate the immunological processes that lead to type I diabetes (DM-I). It was subsequently suggested that A1 beta-casein may also be a risk factor for coronary heart disease (CHD), based on between-country correlations of CHD mortality with estimated national consumption of A1 beta-casein in a selected number of developed countries. A company, A2 Corporation was set up in New Zealand in the late 1990s to test cows and market milk in several countries with only the A2 variant of beta-casein, which appeared not to have the disadvantages of A1 beta-casein. The second part of this review is a critique of the A1/A2 hypothesis. For both DM-I and CHD, the between-country correlation method is shown to be unreliable and negated by recalculation with more countries and by prospective studies in individuals. The animal experiments with diabetes-prone rodents that supported the hypothesis about diabetes were not confirmed by larger, better standardised multicentre experiments. The single animal experiment supporting an A1 beta-casein and CHD link was small, short, in an unsuitable animal model and had other design weaknesses. The A1/A2 milk hypothesis was ingenious. If the scientific evidence had worked out it would have required huge adjustments in the world's dairy industries. This review concludes, however, that there is no convincing or even probable evidence that the A1 beta-casein of cow milk has any adverse effect in humans. This review has been independent of examination of evidence related to A1 and A2 milk by the Australian and New Zealand food standard and food safety authorities, which have not published the evidence they have examined and the analysis of it. They stated in 2003 that no relationship has been established between A1 or A2 milk and diabetes, CHD or other diseases.
Now here's Mat "The Kraken" Lalonde, PhD, on someone's Facebook thread a while back.
The whole A2 versus A1 milk issue was a fabrication by New Zealand farmers who wanted to sell more milk fro their A2 producing cows. Anyone with basic chemistry and biochemistry knowledge can cut through the arguments that were provided by the A2 side. The original studies were in vitro and did not use the full spectrum of enzymes that human beings possess for digestion. Dipeptidyl Peptidase IV and Prolyl Endopeptidase have the ability to shred BCM7 to pieces. For more information see: (a) Teschemaker, H.; Umbach, M.; Hamel, U.; Praetorius, K.; Ahert-Hilder, G.; Brantl, V.; Lottspeich, F.; Henschen, A. J. No Evidence for the Presence of b-Casomorphins in Human Plasma After Ingestion of Cow’s Milk or Milk Products. Dairy Res. 1986, 53, 135–138. (b) Hill, J. P.; Crawford, R. A.; Boland, M. J. Milk and Consumer Health: A Review of the Evidence for a Relationship Between the Consumption of Beta Casein A1 with Heart Disease and Insulin Dependent Diabetes Mellitus. Proc. NZ Soc. Animal Production 2002, 62, 111–114. (c) Truswell, A. S. The A2 Milk Case: A Critical Review. European Journal of Clinical Nutrition, 2005, 59, 623–631. (d) Chin-Dusting, J.; Shennan, J.; Jones, E.; Williams, C.; Kingwell, B.; Dart, A. Effect of Dietary Supplementation with b-Casein A1 or A2 on Markers of Disease Development in Individuals at High Risk of Cardiovascular Disease. British Journal of Nutrition. 2006, 95, 136–144
And...
The effects of BMC7 are only manifested when the molecule is injected into human beings or lab animals. They do not manifest when the molecule is ingested. BMC7 is not detected in the guts of human beings when A1 milk is ingested. That is because of what I've mentioned earlier. The in vitro tests originally used to detect and isolate BMC7 did not have the full spectrum of human digestive enzymes. Human beings are fully capable of digesting BMC7. It is true that BMC7 is problematic when isolated from incomplete in vitro digestion and injected into human beings. However, there are no detrimental effects noted when A1 milk or BMC7 is ingested because human beings digest BMC7. Now if your view is that milk is unhealthy, fine. Just don't use BMC7 to justify your position. Milk is calorically dense and not very satiating (liquids do not cause a whole lot of stomach distension), as such, it is easily over consumed. This can lead to weight gain excess calcium and other problems. Add lactose and casein intolerance to the mix and you have plenty of legitimate reasons to avoid dairy. BMC7 is not one of them, however.
En wat zegt Mike erover:
https://www.fatsforum.nl/topic/a1-en-a2-melk-en-caseine#post-2130
https://www.fatsforum.nl/topic/a1-en-a2-melk-en-caseine#post-2163
Learning all the time, Poekie. De boodschap is: niet teveel inzitten over dat A1/A2-verhaal en lekker genieten van rauwmelkse zuivel. Ik weet van verschillende mensen die zich met rauwe melk hebben geheeld van slopende ziektes, met A1-melk welteverstaan.
Mike
ositive comments about A2 Milk by UK Doctor
